Hey Everyone,
I've recently arrived in Dubai on a visit visa for the purpose of exploring UAE as a tourist as well as seeking employment opportunities. I am an ACCA Affiliate and hold a BSc in Applied Accounting from Oxford Brookes (as part of ACCA). Additionally, I've completed a BSc in Computing and Information Systems from Goldsmiths, University of London. I have professional fluency in English and experience in web development and consulting, gained through freelancing.
Despite not having specific experience in Accounting or Finance, I'm open to entry-level positions from registered companies in the UAE. My interests lie in areas like Web Development, Software Engineering, Audit or Consulting (preferably in the Big 4), Finance, and Management Consulting.
If anyone could assist me in finding a permanent job within these fields, I would greatly appreciate your help.
Looking forward to your valuable suggestions.
Thank you so much!

Hey Everyone,
I've recently arrived in Dubai on a visit visa for the purpose of exploring UAE as a tourist as well as seeking employment opportunities. I am an ACCA Affiliate and hold a BSc in Applied Accounting from Oxford Brookes (as part of ACCA). Additionally, I've completed a BSc in Computing and Information Systems from Goldsmiths, University of London. I have professional fluency in English and experience in web development and consulting, gained through freelancing.
Despite not having specific experience in Accounting or Finance, I'm open to entry-level positions from registered companies in the UAE. My interests lie in areas like Web Development, Software Engineering, Audit or Consulting (preferably in the Big 4), Finance, and Management Consulting.
If anyone could assist me in finding a permanent job within these fields, I would greatly appreciate your help.
Looking forward to your valuable suggestions.
Thank you so much!

So tomorrow I need to go sell some clothes and I was bringing the tote out to my car. My car is in the garage so I unlocked the car and set my keys back on the key holder in the house. I bring the tote out and as I'm walking to the car, I hear the car lock itself. Completely in shock, I set the tote down and go to one of the side doors and pull on it and I find the door is locked. I then go to the driver door and pull on it and that door is locked, too. So then I go inside to get my keys and bring them outside to unlock my car and load the tote.
Is this a normal feature for a 2018 Nissan Versa? I'm aware of the auto-lock feature once it hits 15 mph but I've never had it auto-lock with the engine off and key no where near the car.

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Hello Dear community,
I need your help, or, at least I think I do. I've come into a point in the game in which I don't know how to go further. I'll explain; I have gear level 32 (with my special copper mace equipped). I play in a PvE singleplayer world with me myself and I. At this moment, I've killed several V blood targets;

• Alpha wolf
• Rufus the foreman
• Keely the frost archer
• Errol the stonebreaker
• Goreswine the ravanger
• Grayson the armourer
• Lidya the Chaos archer

Right now, when I look at my goals, I need to create a Castle Throne, which needs Iron. Another Goal I can set myself is upgrading the Castle heart, which needs glass.
Now it feels like both Iron and Glass are resources which are way out off my league still, with only an equipment level of 32. However, with some smart play, and using wolf form, I was able to sneak near the Iron mine and harvest some Iron. However, for me to be able to create Iron ingots, I need to kill Quincey the Bandit King, which is level 37, a long way from my level 32.
Any tips on how to proceed? Which steps should I take to have a better shot at getting Glass and Iron? Everything felt balanced up until now, but now I feel like a weak little vampire baby.
Thanks in advance!

Hello
I've recently come across this community and it has compelled me to do a significant amount of self-reflection. It didn't necessarily offer me new revelations, but it provided the final push I needed to acknowledge an inconvenient truth: I have been wasting my time gaming.
I have struggled with depression this past school year, and I've used video games as a crutch, despite knowing they don't provide any real relief. Too often, I've found myself neglecting my studies to play games for hours on end. The most troubling part is that I'm not even enjoying the time I spend gaming. There's a constant nagging thought in the back of my mind telling me I'm wasting precious time and falling short of my potential.
However, what really lit a fire under me and sparked my desire to quit gaming entirely was my girlfriend. We've been together for nearly six years, and she's truly phenomenal. We share dreams of a future together and she does so much to help me succeed and reduce my stress. This allows me to focus on my university studies and work towards securing a job that can support us both. But when I play video games, I can't help but feel I'm not holding up my end of the bargain. It's struck me hard: why am I wasting time on games when I could be advancing my studies and securing our future?
I am resolved to change, not only for my own sake but also for the love of my life. I want to give her everything she deserves and more, and I know I can't achieve that by squandering hours each day on something that ultimately doesn't matter.
I am truly grateful to have found this subreddit. Moving forward, I'm considering posting regular accountability updates here.
Sorry if this post seems a bit scattered. I've spent most of the last day gaming, it's almost 4 am now, and I'm dreadfully aware of how much I've set myself back for tomorrow. But, this serves as a reminder of why I must change. And I am committed to doing so, for myself and for the woman I love.

I sell collectibles on eBay as a side hustle, I don’t spend any of the money that comes from sales on myself, every penny just gets reinvested and spent on more inventory. I’m hoping to grow it into something that will pay me income hopefully within the next 5-8 years. If I don’t have money in my side hustle account, I don’t buy more inventory. I’m very disciplined when it comes to spending. My credit score is almost perfect and I have no other debt than my mortgage and student loan. Would it be a good idea to get a credit card to make these purchases on for points/benefits etc? I could just make a standing order from my side hustle account to pay it off in full each month. I’m not a registered business but I do self assessment tax returns each January. Any advice or pointers on specific cards (if it’s a good idea) would be much appreciated.

Hello fellows, familia. Grab a coffee/joint/gamersupp.
This is just a public health awareness thread about some little known things amongst the normans. Reddit wont let me post links, but what I will do is post study title and authors/year, and some cases abstract copy paste. I will do at the bottom after the statements.
So, as you all know, COVID is a thing still, and many didn't actually know what it was capable of, outside of comparisons to other coronaviruses and the acute illness. Right now, acute illness is incredibly manageable, but the long term effects on organs is becoming alarming, even in mild cases. So lets take it from the top, so we are on the same page. We will start on organ systems and virus character.
So, covid is pretty much a vascular virus, which seems to disrupt vasculature. Lets start before vaccines to demonstrate to those among us suspicious that the vaccine is the cause. (Its fine to worry about tech, i dont judge).
Every person in Sweden diagnosed with COVID-19 from Feb. 1, 2020, to Sept. 14, 2020 — a total of nearly 87,000 people. Their median age was 48, and 57% were women. Researchers compared them with more than 348,000 Swedish people of similar age and sex who did not have the virus.
In the week after a COVID-19 diagnosis, the risk of a first heart attack increased by three to eight times. The risk of a first stroke caused by a blood clot multiplied by three to six times. In the following weeks, both risks decreased steadily but stayed elevated for at least a month.
Thats PRE vax. In a population which probably have the obesity % of the US and a healthy hiking and outdoor culture. US obesity rate is 40%+. Sweden perhaps half or less. Also, viral load on exposure can be a large determination of severity acutely, but perhaps also viral creep inside the body and the resulting dysfunction and damage.
​
Risk of Cardiovascular Events after Covid-19: a double-cohort study Objective To determine absolute and relative risks of either symptomatic or asymptomatic SARS-CoV-2 infection for late cardiovascular events and all-cause mortality.
Conclusions Either symptomatic or asymptomatic SARS-CoV-2 infection is associated with increased risk of late cardiovascular outcomes and has causal effect on all-cause mortality in a late post-COVID-19 period
Characteristics of a COVID-19 Cohort With Large Vessel Occlusion: A Multicenter International Study

CONCLUSION:

COVID-19 was an independent predictor of incomplete revascularization and poor outcomes in patients with stroke due to LVO. Patients with COVID-19 with LVO were younger, had fewer cerebrovascular risk factors, and suffered from higher morbidity/mortality rates.
Long-term cardiac pathology in individuals with mild initial COVID-19 illness
In this cohort of selected previously well and home-isolating individuals initially evaluated at a median of 109 days and followed-up at 329 days after diagnosis of COVID-19 infection, respectively, the ongoing cardiac symptoms were related to mild but notable imaging findings, suggesting inflammatory cardiac involvement. At baseline, participants with cardiac symptoms had higher mapping values, suggesting diffuse myocardial inflammation, and more frequent pericardial enhancement, suggesting pericardial inflammatory involvement compared to asymptomatic participants. At follow-up, 53% of the cohort had persistent cardiac symptoms, whereas new symptoms developed in 5%.
Transcriptomic profiling of cardiac tissues from SARS-CoV-2 patients identifies DNA damage
In this study, we investigated the host transcriptome landscape of cardiac tissues collected at rapid autopsy from seven SARS-CoV-2, two pH1N1, and six control patients using targeted spatial transcriptomics approaches. Although SARS-CoV-2 was not detected in cardiac tissue, host transcriptomics showed upregulation of genes associated with DNA damage and repair, heat shock, and M1-like macrophage infiltration in the cardiac tissues of COVID-19 patients. The DNA damage present in the SARS-CoV-2 patient samples, were further confirmed by γ-H2Ax immunohistochemistry.
In comparison, pH1N1 showed upregulation of interferon-stimulated genes, in particular interferon and complement pathways, when compared with COVID-19 patients. These data demonstrate the emergence of distinct transcriptomic profiles in cardiac tissues of SARS-CoV-2 and pH1N1 influenza infection supporting the need for a greater understanding of the effects on extra-pulmonary organs, including the cardiovascular system of COVID-19 patients, to delineate the immunopathobiology of SARS-CoV-2 infection, and long term impact on health.
​

Great article by British Heart Foundation.

Is coronavirus a disease of the blood vessels?

Updated 12 May 2023
Dr Phoebe Kitscha explores how and why Covid-19 affects the whole circulatory system, and the research that is trying to tackle it.
​
March 27, 2020

Potential Effects of Coronaviruses on the Cardiovascular SystemA Review

'Coronavirus disease 2019 is associated with a high inflammatory burden that can induce vascular inflammation, myocarditis, and cardiac arrhythmias. Extensive efforts are underway to find specific vaccines and antivirals against SARS-CoV-2. Meanwhile, cardiovascular risk factors and conditions should be judiciously controlled per evidence-based guidelines.'
​
2022
Long-term cardiovascular outcomes of COVID-19
our results provide evidence that the risk and 1-year burden of cardiovascular disease in survivors of acute COVID-19 are substantial. Care pathways of those surviving the acute episode of COVID-19 should include attention to cardiovascular health and disease.
Acute and postacute sequelae associated with SARS-CoV-2 reinfection
The evidence shows that reinfection further increases risks of death, hospitalization and sequelae in multiple organ systems in the acute and postacute phase. Reducing overall burden of death and disease due to SARS-CoV-2 will require strategies for reinfection prevention.
2020
Long covid: Damage to multiple organs presents in young, low risk patients
Initial data from 201 patients suggest that almost 70% had impairments in one or more organs four months after their initial symptoms of SARS-CoV-2 infection.
SARS-CoV-2 infection and persistence in the human body and brain at autopsy
We show that SARS-CoV-2 is widely distributed, predominantly among patients who died with severe COVID-19, and that virus replication is present in multiple respiratory and non-respiratory tissues, including the brain, early in infection. Further, we detected persistent SARS-CoV-2 RNA in multiple anatomic sites, including throughout the brain, as late as 230 days following symptom onset in one case. Despite extensive distribution of SARS-CoV-2 RNA throughout the body, we observed little evidence of inflammation or direct viral cytopathology outside the respiratory tract. Our data indicate that in some patients SARS-CoV-2 can cause systemic infection and persist in the body for months.
​

Endothelial dysfunction in COVID-19: an overview of evidence, biomarkers, mechanisms and potential therapies

SARS-CoV-2 infection primarily affects the pulmonary system, but accumulating evidence suggests that it also affects the pan-vasculature in the extrapulmonary systems by directly (via virus infection) or indirectly (via cytokine storm), causing endothelial dysfunction (endotheliitis, endothelialitis and endotheliopathy) and multi-organ injury.
Mounting evidence suggests that SARS-CoV-2 infection leads to multiple instances of endothelial dysfunction, including reduced nitric oxide (NO) bioavailability, oxidative stress, endothelial injury, glycocalyx/barrier disruption, hyperpermeability, inflammation/leukocyte adhesion, senescence, endothelial-to-mesenchymal transition (EndoMT), hypercoagulability, thrombosis and many others. Thus, COVID-19 is deemed as a (micro)vascular and endothelial disease

Lingering cardiac involvement in previously well people after mild COVID-19

The mild but persistent non-ischemic cardiac inflammation that we describe in this study was not associated with overt structural heart disease or troponin release. Although it is triggered by a viral infection, profound myocardial injury or functional impairment is not typical, contrary to the classical definition of viral myocarditis2. Its pathophysiology is more reminiscent of findings in other chronic diffuse inflammatory syndromes that occur post-virally (for example, human immunodeficiency virus–associated cardiomyopathy)3or as a result of autoimmunity (for example, systemic lupus erythematosus4,5). In these cases, persistent subclinical cardiovascular inflammation seems to predispose people to a poor prognosis and the development of heart failure. Non-ischemic cardiac inflammatory involvement is therefore emerging as an important risk factor, and the long-term prognostic relevance of post-acute COVID-19 cardiac involvement in previously healthy people with mild initial COVID-19 illness requires further investigation.

​
July 27, 2020

Outcomes of Cardiovascular Magnetic Resonance Imaging in Patients Recently Recovered From Coronavirus Disease 2019 (COVID-19)

Conclusions and Relevance In this study of a cohort of German patients recently recovered from COVID-19 infection, CMR revealed cardiac involvement in 78 patients (78%) and ongoing myocardial inflammation in 60 patients (60%), independent of preexisting conditions, severity and overall course of the acute illness, and time from the original diagnosis. These findings indicate the need for ongoing investigation of the long-term cardiovascular consequences of COVID-19.
​

Post-COVID-19 Syndrome

Kamath, Vasantha; Anand, R.1; Radhakrishnan, Buvana1; Markanday, Kushal1
Post-COVID-19 “recovery” cannot be gauged solely on a negative polymerase chain reaction or hospital discharge. There is remarkable variation in the duration, severity, and fluctuation of symptoms, which can affect survivors’ quality of life, functional status, cognition, and mood, and lead to severe disability. Given the global scale of this pandemic, it is apparent that the health-care needs for patients with sequelae of COVID-19 will continue to increase for the foreseeable future.
​
Immune system deviations?
We heard initial outbreaks of fungal infections of various types. Yes any infection can cause reduction in immune cells. Either because the immune system is busy diverted into tissues, fighting infection, or from apoptosis etc.
The deposits of antigens or viral pieces in many parts of the body, is perhaps causing problems, but also there appears to be markers of T cell exhaustion in some with high PD1 expression on some important immune cells too?
Long COVID following mild SARS-CoV-2 infection: characteristic T cell alterations and response to antihistamines Patients with long-COVID had reduced CD4+ and CD8+ effector memory (EM) cell numbers and increased PD-1 (programmed cell death protein 1) expression on central memory (CM) cells, whereas the asymptomatic participants had reduced CD8+ EM cells only and increased CD28 expression on CM cells. 72% of patients with long COVID who received HRA reported clinical improvement
The risk of pancreatic adenocarcinoma following SARS-CoV family infection
Our findings suggest that pancreatic adenocarcinoma is the most probably malignancy happening after infection with SARS-CoV family
The SARS-CoV-2 E protein induces Toll-like receptor 2-mediated neonatal lung injury in a model of COVID-19 viremia that is rescued by the glucocorticoid ciclesonide
This study provides insight into the pathogenesis of ALI and alveolar remodeling with SARS-CoV-2 viremia in children, whereas revealing the efficacy of steroids.
A new case-control study of Brazilian healthcare workers(HCWs) suggests as many as 27% developed long COVID after infection, and multiple infections raised the risk. The findings were published today in Infection Control & Hospital Epidemiology.
Estimates of the prevalence of long COVID, defined by the Centers for Disease Control and Prevention as new, returning, or lasting symptoms persisting 4 or more weeks after initial COVID-19 infection vary, with some studies showing as many as 43% of infected people will have some lingering symptoms 1 month after COVID-19 confirmation.
​
"39 (58%) patients (with glioma) experienced tumor progression following COVID-19 infection at a median of 34 days after testing positive for COVID-19.. 22 patients had measurably increased tumor area by a median of 63%; 18 of which constituted hyperprogression; 16 patients developed multifocal disease; 8 developed new nodular enhancement; 3 developed leptomeningeal disease (LMD); and 2 experienced increased infiltrative disease alone. 10 patients’ presentation with new glioma was preceded by COVID-19 infection by a median of 31 days. GBM (glioblastoma) patients represented the majority of progression events, among whom 59% progressed within 60 days of documented infection.. Glioma patients appear to have disease progression at an accelerated pace in the first two months after COVID-19 infection."
​
SARS-CoV-2 infection: The role of PD-1/PD-L1 and CTLA-4 axis
The most important diagnostic feature in individuals with COVID-19 is lymphocyte depletion, most importantly, T-cells. Due to the induction of interferon-γ (INF-γ) production by neutrophils and monocytes, which are abundantly present in the peripheral blood of the individuals with COVID-19, the expression of inhibitory immune checkpoints including, PD-1 (programmed death), PD-L1 and CTLA4 on the T-cells' surface is enhanced. The purpose of this review is to discuss the functions of these checkpoints and their effects on the dysfunction and exhaustion of T-cells, making them almost ineffective in individuals with COVID-19, especially in the cases with extreme symptoms.
​
COVID-19 and renal involvement: a prospective cohort study assessing the impact of mild SARS-CoV-2 infection on the kidney function of young healthy males
Mild COVID-19 is associated with mild renal involvement without AKI. Changes in the renal function appear to be related to reduced creatinine clearance and possible albumin leakage in the acute phase of the disease. The reduction in creatinine clearance is not predicted by viral load, and it appears to be a long-term effect of the disease that can last for at least 6 months.
​
Reduction and Functional Exhaustion of T Cells in Patients With Coronavirus Disease 2019 (COVID-19)
Conclusions: T cell counts are reduced significantly in COVID-19 patients, and the surviving T cells appear functionally exhausted. Non-ICU patients with total T cells counts lower than 800/μL may still require urgent intervention, even in the immediate absence of more severe symptoms due to a high risk for further deterioration in condition.
​
Brain
​
Cerebral blood flow in patients recovered from mild COVID-19
In this study, lower WM CBF, as well as widespread regional CBF changes identified using quantitative MRI, was found in mild COVID-19 patients. Further studies are needed to determine the reliability of this newly identified COVID-19 brain imaging marker and determine what drives these CBF changes.
​
REVIEW

Alzheimer’s disease risk after COVID-19: a view from the perspective of the infectious hypothesis of neurodegeneration

Olivera, Eugenia; Sáez, Albany; Carniglia, Lila; Caruso, Carla; Lasaga, Mercedes; Durand, Daniela*
The present article aimed at reviewing the most recent literature supporting the infectious hypothesis of AD, and addressed the probable risk of developing Alzheimer-like dementia after severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection, based on evidence linking viral infections and amyloidosis.
​

• EWS
• 10 February 2022

Heart-disease risk soars after COVID — even with a mild case

Massive study shows a long-term, substantial rise in risk of cardiovascular disease, including heart attack and stroke, after a SARS-CoV-2 infection.
JOURNAL ARTICLE

Global Prevalence of Post-Coronavirus Disease 2019 (COVID-19) Condition or Long COVID: A Meta-Analysis and Systematic Review

This study finds post-COVID-19 condition prevalence is substantial; the health effects of COVID-19 seem to be prolonged and can exert stress on the healthcare system.
Review articleBrain disorders: Impact of mild SARS-CoV-2 may shrink several parts of the brain
Coronavirus (COVID-19) is a highly infectious respiratory infection discovered in Wuhan, China, in December 2019. As a result of the pandemic, several individuals have experienced life-threatening diseases, the loss of loved ones, lockdowns, isolation, an increase in unemployment, and household conflict. Moreover, COVID-19 may cause direct brain injury via encephalopathy. The long-term impacts of this virus on mental healthand brain function need to be analysed by researchers in the coming years. This article aims to describe the prolonged neurological clinical consequences related to brain changes in people with mild COVID-19 infection. When compared to a control group, people those who tested positive for COVID-19 had more brain shrinkage, grey matter shrinkage, and tissue damage. The damage occurs predominantly in areas of the brain that are associated with odour, ambiguity, strokes, reduced attention, headaches, sensory abnormalities, depression, and mental abilities for few months after the first infection. Therefore, in patients after a severe clinical condition of COVID-19, a deepening of persistent neurological signs is necessary.
​
continuing ...

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Contact me to get Iman Gadzhi - Agency Navigator by chatting me on +44 759 388 2116 on Telegram/Whatsapp.
I have Iman Gadzhi - Agency Navigator.
Iman Gadzhi - Agency Navigator course is one of the best products on how to start a marketing agency.
Iman Gadzhi - Agency Navigator includes over 50 hours of step-by-step training covering EVERY aspect of building an agency from scratch. This is almost a plug & play system with enough success stories to back it up! Signing clients, running Facebook ads, building out your team, on-boarding clients, invoicing, sales... this course has everything covered for you.
The topics inside Iman Gadzhi - Agency Navigator course include:

1. Agency Navigator course Core Curriculum
   
2. Custom E-Learning Platform For Agency Owners
   
3. Financial Planner, Revenue Calculator, Outreach Tracker & More Tools
   
4. Websites Templates, Funnels, Ads & More
   
5. Template Contracts, Sales Scripts, Agreements & More
   

The lessons in Iman Gadzhi - Agency Navigator will teach you how to:
- Starting Your Agency
- Finding Leads
- Signing Clients
- Getting Paid
- Onboarding Clients
- Managing Client Communication...
...and much, much more!
To get Iman Gadzhi - Agency Navigator contact me on:
Whatsapp/Telegram: +44 759 388 2116
Reddit DM to u/RequestCourseAccess
Email: silverlakestore[@]yandex.com (remove the brackets)

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Hello hello :) I'm going to try this again! I am looking to add more people to my snail-mail pen pal list!
A little bit about me:
I live in the Midwest (USA) with a huge passion for writing letters and photography
I absolutely LOVE reading (romance, social science, and poetry are my top genres)
I work as a clinical coach at a substance abuse rehabilitation treatment facility and will be obtaining my substance abuse counselor certification in the near future
I graduated college in 2022 with a BA in psychology and a BA in social and criminal justice
And now for some of my favorites:
Artist: Taylor Swift
Band: EXES
Color: Blue
Animal: Penguin
Food: Pasta
I have no preference on age/gendelocation! If you want to become pen pals, SWEET! Feel free to comment or DM me :) I can't wait to get to writing to more people!

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Read here : https://casadeemontessori.blogspot.com/2023/06/discover-perfect-daycare-near-you-casa.html
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Hello, nearly fully diagnosed autistic + possibly have unmedicated ADHD ( plus, suffering from depression )
At school, I've always been the kid that doesn't works at all, only aiming for the minimum to pass to the next year. Then, I got into college within a prestigious political sciences school. 5 years later, I've failed two separate years for mostly external reasons, and they've kicked me out some months ago.
Right now, I am doing a journalism internship that I was supposed to do sponsored by my school.
My problem is, I'm genuinely afraid on what to do, where to go, from this ? The job is OK, I'm good at it ( It's mostly being on the computer searching things ) but I hate having to go to the office each days, having to talk to coworkers, saying " Hello ! :) " to each one that I come across in corridors. Having unnecessary conversations, etc, etc.
After a workday, I feel like I'm spending most of the afternoon and night charging up for the next day, and that within weekends I have to choose between going out to do things, see friends, or stay at home to not be burnt-out on Monday.
And it's only been a month. I genuinely can't see how I can do that for the rest of my life.
I'm currently within a turning point in my life, after that internship I'll have to choose between continuing studies or pushing the journalism career and I'm scared. I don't see what jobs could allow me to stay healthy while still having a decent pay. I think that I have a lot of passions, and competences, but the context in which I have to use them depresses me.
What are you all doing as a job ? I'm really curious on how you are coping with all the things that I've mentioned. I'm seeing lots of IT folks but I'm the worst on maths lol.I'm desperate to the point that I'm unironically thinking of just finishing my diagnosis and living on welfare money forever, or try to start a youtube channel on my special interests and get a paid patreon out of it. Thank you all.

My student who is studying vehicle engineering in China needs some help with his future career path, he is busy applying to different universities abroad and mainly trying to get into Bologna, Italy. He wanted me to reach out to some community groups and ask for advice on the path with the most career opportunities/highest likelihood of finding a job. Any guidance would be appreciated. He wanted to know which of these study paths would be best. The fields of study he needs to decide between were the following:

1. Electronic studies of the car
   
2. Power and transmission
   
3. Vehicle Intelligence (Statistics and internal communication / Dynamics control / Electro magnetic compatibility)
   

Contact me to get Iman Gadzhi - Agency Navigator by chatting me on +44 759 388 2116 on Telegram/Whatsapp.
I have Iman Gadzhi - Agency Navigator.
Iman Gadzhi - Agency Navigator course is one of the best products on how to start a marketing agency.
Iman Gadzhi - Agency Navigator includes over 50 hours of step-by-step training covering EVERY aspect of building an agency from scratch. This is almost a plug & play system with enough success stories to back it up! Signing clients, running Facebook ads, building out your team, on-boarding clients, invoicing, sales... this course has everything covered for you.
The topics inside Iman Gadzhi - Agency Navigator course include:

1. Agency Navigator course Core Curriculum
   
2. Custom E-Learning Platform For Agency Owners
   
3. Financial Planner, Revenue Calculator, Outreach Tracker & More Tools
   
4. Websites Templates, Funnels, Ads & More
   
5. Template Contracts, Sales Scripts, Agreements & More
   

The lessons in Iman Gadzhi - Agency Navigator will teach you how to:
- Starting Your Agency
- Finding Leads
- Signing Clients
- Getting Paid
- Onboarding Clients
- Managing Client Communication...
...and much, much more!
To get Iman Gadzhi - Agency Navigator contact me on:
Whatsapp/Telegram: +44 759 388 2116 (Telegram: multistorecourses)
Reddit DM to u/CourseAccess
Email: silverlakestore[@]yandex.com (remove the brackets)